A
REFERENCES CITED:

1. McMullin, T., et al., Evidence that atrazine and diaminochlorotriazine inhibit the estrogen/progesterone induced surge of Luteinizing Hormone in female Sprague-Dawley rats without changing estrogen receptor action. Toxicol. Sci., 2004. 79: p. 278-286.

2. Narotsky, M., et al., Strain comparisons of atrazine-induced pregnancy loss in the rat. Reprod. Toxicol., 2001. 15(1): p. 61-69.

3. Cummings, A., B. Rhodes, and R. Cooper, Effect of atrazine on implantation and early pregnancy in 4 strains of rats. Toxicol. Sci., 2000. 58(1): p. 135-143.

4. Cooper, R.L., T.E. Stoker, and W.K. McElroy, Atrazine (ATR) disrupts hypothalamic catecholamines and pituitary function. The Toxicologist, 1999. 42: p. 60-66.

5. Cooper, R.L., et al., Atrazine disrupts the hypothalamic control of pituitary-ovarian function. Toxicol. Sci., 2000. 53: p. 297-307.

6. Stoker, T.E., C.L. Robinette, and R.L. Cooper, Maternal exposure to atrazine during lactation suppresses suckling-induced prolactin release and results in prostatitis in the adult offspring. Toxicol. Sci., 1999. 52: p. 68-79.

7. Rayner, J., C. Wood, and S. Fenton, Exposure parameters necessary for delayed puberty and mammary gland development in Long–Evans rats exposed in utero to atrazine. Toxicol. Appl. Pharmacol., 2004. 195(23-34).

8. Rayner, J., R. Enoch, and S. Fenton, Adverse effects of prenatal exposure to atrazine during a critical period of mammary gland growth. Toxicol. Sci., 2005. 87(1): p. 255-266.

6. Atrazine causes pregnancy loss, delayed development, and low birth- weight in laboratory rodents
AA